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A can directly disrupt the ETC and interfere with various mitochondrial matrix proteins and putative components of the mPTP, ultimately resulting in mitochondrial dysfunction (77-80)
Alternative splicing of IGF-I pre-mRNA can produce three multiple mRNA species, depending on the inclusion of alternative leader and C-terminal exons [2]
Various components of injured or dying cells including cytosol, mitochondria, and nucleus release DAMPs by the process of necrotic, pyroapoptotic, and necroapoptotic cell death which contribute to inflammation (26, 27)
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