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To overcome these limitations, herein, the tumor-selective transport of a physiological stable ruthenium-based catalyst for the oxidation of GSH into GSSG as a nanoreactor is proposed

mTORC1 drives HIF-1 and VEGF-A signalling via multiple mechanisms involving 4E-BP1, S6K1 and STAT3

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Eicosanoids are not stored, and they have an extremely short half-life, being rapidly metabolized to inactive products