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Importantly, CHAC2 did not localize to the mitochondria irrespective of extracellular cystine availability, further suggesting divergent roles for these proteins in mediating intracellular GSH catabolism

Evidence is mixed: some animal NAFLD models favor vitamin C, whereas human NASH trials (e.g., PIVENS) showed histologic improvement with high-dose vitamin E, albeit with limited impact on fibrosis

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[] Notably, ozones dual identity toxic pollutant at high doses, therapeutic agent at low controlled doses creates a mechanistic paradox that is both conceptually intriguing and clinically contentious