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Several mechanisms of action of NAC have been proposed across different cardiovascular diseases, including enhancement of NO metabolism by supplying free sulfhydryl groups needed for the activation of guanylate cyclase, inhibition of the angiotensin-converting enzyme, prevention of the oxidation of nitrate compounds, reduction of ROS and other free radicals, anti-inflammatory activity, and antiplatelet effects [9, 172, 173]

Berberine ameliorates iron levels and ferroptosis in the brain of 3 Tg-AD mice

LSD1 produces hydrogen peroxide in response to DNA damage

This mechanism not only promotes epithelial ferroptosis but also drives macrophage M1 polarization [48]

this enables STAT3 translocation to the nucleus and transcriptional initiation of target genes, including suppressor of cytokine signalling 3 (SOCS3), which is an inhibitor of LepRbJAK2 signalling