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Together, these data support the notion that stress-induced lifespan extension is likely a result of a cell-wide coordinated response, alongside robustly activated canonical HSR

Dynamic O-GlcNAcylation coordinates ferritinophagy and mitophagy to activate ferroptosis

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To overcome the limitations of poor absorption, various delivery systems such as nanoemulsions, liposomes, solid lipid nanoparticles (SLNs), and inclusion complexes with cyclodextrins have been developed to enhance bioaccessibility and stability [5]

Knockdown of mitophagy (a specific cellular process for elimination of dysfunctional mitochondria by autophagosome engulfment for subsequent lysosomal degradation) genes also reduced heat stress resistance in worms, due to increased protein aggregate formation caused by autophagy downregulation [118]