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In addition, by activating the PI3K/Akt pathway and speeding up the translocation of NFE2-Nrf2 to the nucleus via KEAP1 degradation, hASC-EV treatment significantly decreased oxidative damage

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Alpha-Synuclein physiology and pathology: a perspective on cellular structures and organelles

In vitro activity of N-acetylcysteine against Stenotrophomonas maltophilia and Burkholderia cepacia complex grown in planktonic phase and biofilm

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