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This is consistent with the theory of lack of cyclic expression of HOXA10 in the mid-luteal phase, as hypermethylation of the HOXA10 promoter could suppress its expression

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32 , 33 Another therapeutic strategy approach focuses on targeting crystallin aggregation, 34 with oxysterols such as LAN and 25-hydroxycholesterol emerging as promising candidates for preventing or reversing protein aggregation in cataracts
