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In fact, GSH in the plasma is maintained at very low concentration because of the metabolism of GSH by many other cells (Sies and Graf, 1985

There is growing evidence, including findings obtained in experiments using heterochronic parabiosis in mice as a model system, that age-related changes in circulating anti-geronic factors importantly contribute to the regulation of vascular aging processes and the genesis of endothelial dysfunction and oxidative stress in a noncell autonomous manner [9]

Cardiac and neurochemical biomarkers were also significantly altered in hyperthyroid-induced rats compared to the controls

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