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Description
Ageing Res Rev, 72:101503
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Sharma P, Srivastava P, Seth A, Tripathi PN, Banerjee AG, Shrivastava SK
Fact: As we discussed, formulation matters
We therefore hypothesized that the partnership between PIF4 and other PIFs was the reason that PIF4 variants with weakened PIF4 TAD activity (e.g., mTAD and mWFYL) or DNA-binding (mB and RRQQ) could still largely complement the pif4 null mutant (Fig