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In addition, T2D is the result of a complex interaction of environmental and genetic factors, being difficult to establish the role of each one in the pathogenesis of diabetes and in the levels of IGF-1

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MOTS-C activates AMPK pathways to improve insulin sensitivity, mitochondrial function, and metabolic flexibility

10.1002/pro.3798 148

This novel mode of cell death involves the interaction of copper with lipoacylated proteins within the tricarboxylic acid cycle, leading to proteinotoxic stress and culminating in cell death