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J Pathol 241(3):337349

doi: 10.1016/j.preteyeres.2003.12.001

Moreover, NPC1 cell models have shown an inefficient activity of antioxidant enzymes and mitochondrial dysfunction, both of which can contribute to the pathogenesis of the disease [128]

Together, these data show that BMS, which has already been used in multiple phase III clinical trials (NCT03661320, NCT03329846), is not only a potent IDO1 inhibitor, but also displays a significant off-target effect by inhibiting the Q-site of complex I and thereby the main entry of electrons into the respiratory chain

This provides a reasonable stability and delivery strategy