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In vitro tests have also shown a potential role for ROS in induction of adaptive immune responses (through T lymphocytes maturation and activation) and airway hyperresponsiveness (promoting proliferation and contraction of smooth muscle cells) [4,5,91]

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Newer evidence points to additional cellular and physiological effects caused by lack of cellular oxidants and accumulation of excess reducing equivalents, including changes in protein disulfide bond formation, diminished mitochondrial function, and decreased cellular metabolism

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