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LPL (lipoprotein lipase), HL (hepatic triglyceride lipase), and EL (endothelial lipase) are a family of enzymes that operate to remove TG from VLDL and chylomicrons ( 4.2 Lipoprotein lipase Lipoprotein lipase, which catalyzes the release of FA for storage by adipose tissue or oxidation as an energy source by muscle, is the main enzyme in the intravascular breakdown of TG in circulating TRL ( Figure 3 )

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doi: 10.15252/embj.201591973 65 LiangSGuoHMaKLiXWuDWangYet al

This deficiency might contribute to symptoms such as hyperactivity, irritability, and sensory processing issues

The physiological role of PNPLA3 is not completely understood, but a polymorphism in the protein is the predominant genetic risk factor for hepatic steatosis (discussed later)