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Each of these dysfunctional synaptic-related functions due to oxidatively modified key proteins is observed in AD and MCI brains, and we propose that these oxidatively modified proteins contribute to the cognitive dysfunction and related clinical observations in AD and MCI

In the future, it is necessary to combine Mendelian randomization studies to verify causal hypotheses and avoid the confounding factors and potential causal inversion that observational studies cannot avoid

Previously reported DMSO effects on oocytes and embryos are limited to changes in gene expression in animal models 27,33,34,35,36

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