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LncRNA NEAT1 promotes Brutons tyrosine kinase (BTK) transcription by downregulating the transcription factor Krppel-Like Factor 4 (Klf4), which subsequently leads to the activation of the NF-B pathway, NLRP3 inflammation and M1 polarization in BMDMs, whereas lncRNA NEAT1 from endothelial cells enhances M2 polarization via DDX3X/NLRP3 axis (239, 240)

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11/12 patients who received intra-articular BPC-157 for knee pain reported significant relief