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These findings suggest that CPT1C is a regulator of FA homeostasis in cancer cells and might be indirectly involved in the modulation of CPT1A activity through the lowering of cellular malonyl-CoA levels

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10.1111/mmi.15151 72

Mechanistically, hyperoside inhibits fatty acid and triglyceride synthesis via the FXRSCD1/SREBP1 axis, while independently suppressing ATP-citrate lyase (ACLY), thereby reducing acetyl-coenzyme A availability at its source and synergistically attenuating lipid accumulation (58)