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Bhatt DL, Steg PG, Brinton EA, Jacobson TA, Miller M, Tardif JC, Ketchum SB, Doyle RT Jr, Murphy SA, Soni PN, Braeckman RA, Juliano RA, Ballantyne CM

The therapeutic use of PE in GBS is theoretically justified by the need to remove neuro-toxic inflammatory agents that cause the condition following preceding infection, and mechanisms of molecular mimicry, established in axonal forms, as described above

According to Cant et al., Cell Metabolism , increased NAD stimulates PGC-1 activity, driving mitochondrial biogenesis and substrate flexibility

These drawbacks highlight the necessity for more consistent reporting of results and improved coordination between preclinical and clinical evidence streams

However, some people may be at greater risk for Long COVID than others