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Treatment of Wilson's Disease: The purpose of treatment is to eliminate excess copper and prevent re-accumulation

These different results could be accounted for by both the expression of NADPH oxidase induced by KRAS G12D overexpression, which leads to ROS generation, and an increase in NRF2-dependent ROS scavenging in cells expressing physiological levels of KRAS G12D

Dipeptidyl peptidase-4 impairs insulin signaling and promotes lipid accumulation in hepatocytes

Hybrid Polycaprolactone/Alginate Scaffolds Functionalized With VEGF to Promote De Novo Vessel Formation for the Transplantation of Islets of Langerhans