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Here, we show that upon heat and ER stresses HSF-1 is required for the activation of UPR ER in C
The actual directionality of this interaction is still not clear, however the immune response to an external pathogen could provoke unblinded oxidative stress which in turn perpetuates a steady inflammatory cascade (Maes et al
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Surfactant protein C G100S mutation causes familial pulmonary fibrosis in Japanese kindred
