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Rapid proliferation requires the massive synthesis of new proteins, nucleic acids (RNA and DNA), and phospholipids, and the synthesis and modification processes of these substances also require the support of methylation reactions (35)

ENU induces O-alkylguanine lesions in DNA, which are recognized by the MMR system but are not repairable

[] In contrast to Fndc5, myonectin (CTRP15, also a myokine) activates mTOR to suppress autophagy in the liver, [] and CTRP15 deletion promotes fat storage in the adipose tissue and reduces hepatic steatosis

Scientific insights Research on vitamin B12 and fatty liver disease is ongoing and changing

Angiotensin II: role in skeletal muscle atrophy