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The effects of melatonin on mitochondria may be mediated via MT1/2 receptors, resulting in decreased ROS generation, higher antioxidant capabilities, and therefore, in less neural apoptosis, activating nuclear factor erythroid 2-related factor 2, as shown in preclinical models (24,92,103,104)

Given the scarcity of high-quality human trials, peptide injections remain an experimental therapy for musculoskeletal conditions

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Johnson et al., 2018