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ERK5 is a two-kinase transcription factor, and phosphorylation of ERK5 S496 directly inhibits NRF2 protein activity through SUMOylation modification at site 518, an inhibitory effect that can lead to inflammation and mitochondrial dysfunction in macrophages [133]
Helped with tendon pain and overall joint comfort
By modulating mitochondrial function, oxidative stress, and neuroinflammation, KD may exert preventive effects by enhancing metabolic resilience during earlier phases of disease progression and contribute to symptom management in established neurodegenerative conditions [18,19,20]
J Allergy Clin Immunol 154(2):412423
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