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Overexpression of GPX1 attenuates 6-OHDA induced toxicity to dopamine neurons in rodent models of PD [11, 12]
Coupled with the potential of self-perpetuating damage through neuronal atrophy and neuroinflammation, the oxidative stress mechanisms are plausible in the pathophysiology of many brain-related diseases [4]
Related links NCI Pediatric Preclinical Testing Consortium: SARC Clinical trials: Rights and permissions About this article Cite this article Grnewald, T.G.P., Cidre-Aranaz, F., Surdez, D
PAIN, 154(6):874-881
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