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2008 [cited 2022 Sep 3];3

65 This protection is achieved through a variety of biological processes, including interactions with transition metals, neutralization of free radicals, modification of the activity of different enzymes, and impacts on intracellular signaling pathways and gene expression

The oxidation of specific tyrosine and methionine residues in apoA-I contributes to its loss of activity (Shao, 2012)

Anderson EJ, Kypson AP, Rodriguez E, Anderson CA, Lehr EJ, Neufer PD (2009) Substrate-specific derangements in mitochondrial metabolism and redox balance in the atrium of the type 2 diabetic human heart

Similarly, addition of acetyl-CoA, the natural product of DLAT which binds to the same site, also protected PDHC from inhibition by SNO-CoA in a dose-dependent manner (Extended Data Fig