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161 A small molecule called divertin, which can bind specifically to the immunoglobulin-like occludin cell adhesion molecule (IgCAM) of epithelial long MLCK1, was shown to prevent cytokine-mediated MLCK1 recruitment to PAMR without interfering with other facilitative mechanisms (e.g., epithelial migration and wound repair)

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Further structural comparison of our FAT-1907 structure to that of the FAT-LD2 complex shows that, overall, the arrangement of the FAT domain was mostly conserved (root-mean-square deviation: 0.603)