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Mitochondrial free Ca2+ levels and their effects on energy metabolism in Drosophila motor nerve terminals

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Two prosthetic meshes were placed sublay

Transcription factors YAP/TAZ, which reside downstream of VEGF/VEGFR-2, are upregulated on stiffer matrices and in disturbed flow, and in vivo inhibition of YAP/TAZ caused decreased atherosclerosis and EC inflammation (83, 84)
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