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Similar content being viewed by others Introduction Triple-negative breast cancer (TNBC) accounts for ~1020% of all breast cancer cases and is characterized by aggressive growth, high recurrence rates, and limited treatment options due to the absence of estrogen receptor (ER), progesterone receptor (PR), and human epidermal growth factor receptor 2 (HER2) [1, 2]

Cantharidin induces apoptosis of human triple negative breast cancer cells through mir-607-mediated downregulation of EGFR

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