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The theoretical cancer concern applies to TB-500 as well, though perhaps to a lesser extent

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10.1007/s00277-021-04397-z

Upon IB degradation, NF-B is released and translocates into the nucleus, where it recruits p300, which in complex with cAMP response element-binding protein acetylates RelA/p65, thereby activating NF-B transcription and enhancing the expression of downstream pro-inflammatory genes (Lawrence 2009)